The article presents results which indicate that the development of nonalcoholic steatosis in patients with diabetes mellitus type 2 is accompanied with different postprandial changes in lipids, which are closely associated with increased liver fat and low levels of serum adiponectin. Predictor of postprandial lipid response is per cent of liver fat. The degree of fat accumulation in the liver affects insulin resistance, hyperinsulinemia, insulin and postprandial values of insulin and C-peptide. It is shown that a high fat content in the liver is associated with postprandial lipemia, which serves as a potential risk factor for cardiovascular disease.
