THE ROLE OF DISORDERS OF LIPID METABOLISM IN THE PATHOGENESIS OF CHRONIC HEART FAILURE IN PATIENTS WITH POSTINFARCTION CARDIOSCLEROSIS, TYPE 2 DIABETES AND OBESITY

Aim. The aim of our study was determination of the role of of lipid metabolism disorders in the pathogenesis of chronic heart failure in patients with postinfarction cardiosclerosis, type 2 diabetes and obesity.

Materials and methods. There were examined 71 patients with postinfarction cardiosclerosis, type 2 diabetes and obesity, which were divided into four groups. The first group included patients with II functional class of chronic heart failure (n=26), the second — patients with III functional class of chronic heart failure (n=22), the third — patients with IV functional class of chronic heart failure (n=23) and control group consisted of practically healthy people (n=35).

The statistical processing of results was performed with the help of software package “Statistica” (StaSoftInc,USA). The values were calculated: the average (M), dispersion, standard deviation, the median (m), accuracy and level of significance (p). The standard programme of correlation analysis with calculation of average arithmetic means was used: M±m, у, and level of accuracy (p). Pearson correlation coefficient was applied to evaluate the interaction stage between the  samples (r).

Results. The level of LDL-C, TG, VLDL, TC and BMI were significantly increased in patients with III FC of chronic heart failure, postinfarction cardiosclerosis, type 2 diabetes and obesity. And level of LDL-C, TG, VLDL, TC and BMI were significantly decreased in patients with IV FC of chronic heart failure, postinfarction cardiosclerosis, type 2 diabetes and obesity. The study showed that in patients with postinfarction cardiosclerosis, type 2 diabetes and obesity the disorders of lipid metabolism were associated with functional class of chronic heart failure.

The role of disorders of lipid metabolism in patients with postinfarction cardiosclerosis, type 2 diabetes and obesity was determined in the pathogenesis of chronic heart failure.